myocardium$51300$ - ορισμός. Τι είναι το myocardium$51300$
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Τι (ποιος) είναι myocardium$51300$ - ορισμός

STATE WHEN SOME SECTION OF THE MYOCARDIUM (CORRESPONDING TO AREA OF A MAJOR CORONARY OCCLUSION) SHOWS A FORM OF CONTRACTILE ABNORMALITY
Stunned myocardium; Neurogenic stunned myocardium

cardiomyopathy         
  • Stained microscopic section of heart muscle in hypertrophic cardiomyopathy
  • Structural categories of cardiomyopathy
  • The arrhythmia, [[ventricular fibrillation]], seen on an ECG
HEART DISEASE AND A MYOPATHY THAT IS CHARACTERISED BY DETERIORATION OF THE FUNCTION OF THE HEART MUSCLE
Cardiomyopathies; Myocardiodystrophy; Myocardium disorder; Obstructive cardiomyopathy; Myocardial degeneration; NICM; Non-ischemic cardiomyopathy
[?k?:d???m??'?p??i]
¦ noun Medicine chronic disease of the heart muscle.
Myocarditis         
  • hematoxylin and eosin]] stain)
  • Lymphocytic myocarditis (white arrow points to a lymphocyte), commonly showing myocyte necrosis (black arrow), seen as hypereosinophilic cytoplasm with loss of striations.
  • Diffuse ST elevation in a young male due to myocarditis and pericarditis
EXTRINSIC CARDIOMYOPATHY THAT IS CHARACTERIZED AS AN INFLAMMATION OF THE HEART MUSCLE
Acute myocarditis; Inflammation of the heart; Rheumatic myocarditis; Viral myocarditis; Myocardiitis; Post-infectious myocarditis; Infectious myocarditis; Myocardial inflammation; Inflammatory cardiomyopathy; Causes of myocarditis
·noun Inflammation of the myocardium.

Βικιπαίδεια

Myocardial stunning

Myocardial stunning or transient post-ischemic myocardial dysfunction is a state of mechanical cardiac dysfunction that can occur in a portion of myocardium without necrosis after a brief interruption in perfusion, despite the timely restoration of normal coronary blood flow. In this situation, even after ischemia has been relieved (by for instance angioplasty or coronary artery bypass surgery) and myocardial blood flow (MBF) returns to normal, myocardial function is still depressed for a variable period of time, usually days to weeks. This reversible reduction of function of heart contraction after reperfusion is not accounted for by tissue damage or reduced blood flow, but rather, its thought to represent a perfusion-contraction "mismatch". Myocardial stunning was first described in laboratory canine experiments in the 1970s where LV wall abnormalities were observed following coronary artery occlusion and subsequent reperfusion.